Adenomas are also seen in glycogen storage disease types Ib and III. Dramatic advances, particularly in magnetic resonance imaging (MRI), have led to novel techniques to noninvasively diagnose and to characterize patterns of diffuse hepatic disease. Change in the sonographic appearance of adenomas over time may reflect malignant degeneration or hemorrhagic necrosis. In the cancer patient, epidural cord compression by bony metastasis is the commonest myelopathic presentation.

It is low in cost, fast, and readily available. With ultrasonography, geographic areas of radiation injury are hypoechoic relative to the remainder of the liver. MRI signal is therefore not visibly altered in the presence of copper and cannot be used to differentiate Wilson’s disease from other diffuse liver diseases. Types III and IV glycogen storage disease can progress to cirrhosis. A proposed scoring system for diagnosing impending pathologic fractures. Iron first accumulates in periportal hepatocytes and then in the lobular hepatocytes, Kupffer cells, and biliary epithelium. These include increased hepatic synthesis of fatty acids (alcohol), decreased hepatic oxidation or utilization of fatty acids (carbon tetrachloride, high-dose tetracycline), impaired release of hepatic lipoproteins, and excessive mobilization of fatty acids from adipose tissue (starvation, steroids, alcohol). CT can also follow the course of antiviral therapy by observing a reduction in lymph node size.


Mirels H. Metastatic disease in long bones. The liver has quite accurately been called the custodian of the Imaging plays a critical role in the diagnosis and treatment of diffuse liver disease by detecting its presence, determining its distribution and severity, and identifying associated complications, such as cirrhosis, portal hypertension, and malignant disease. The attenuation of the liver is diffusely increased secondary to intraparenchymal iron deposition. On MRI, acute hepatitis may show nonspecific findings such as heterogeneous signal intensity, most apparent on T2-weighted sequences, and a heterogeneous pattern of enhancement on arterial-dominant phase spoiled gradient-echo images. It now accounts for 90% of the cases of hepatitis as a result of contaminated blood transfusions before standard screening.
These techniques may also serve as an early and accurate screening tool for asymptomatic but high-risk patients. Hepatic steatosis with concomitant viral hepatitis can accelerate cellular damage and also decreases the effectiveness of antiviral therapies. The previously mentioned differentiating features of focal fat, including characteristic location, lack of contour abnormality, and normal course of the hepatic vessels, are helpful and are only rarely simulated in metastatic tumor lesions. A true mass can be differentiated from focal regions of fat deposition and focal sparing because these occur in characteristic locations and typically have a geographic shape (but may appear nodular), poorly delineated margins, and, importantly, absence of mass effect on blood vessels. In acute viral hepatitis, the liver and spleen are frequently enlarged. Hope this helps.Commonly without symptoms. The ability to assess liver and spleen size and the presence of ascites is limited on physical examination. The posterior aspect of segment IV, a common area to be spared in diffuse fatty infiltration, has been shown to have aberrant direct gastric venous flow when focal fat sparing is present.


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